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IL-1-mediated and IL-17-mediated cutaneous immune response against S. aureus. S. aureus infection of skin results in production of IL-1a, IL-1b, and
IL-17. These pro-inflammatory cytokines induce keratinocyte production of antimicrobial peptides (e.g. b-defensins 2 and 3, cathelicidin, RNase 7) and
cytokines, chemokines, adhesion molecules, and granulopoiesis factors that promote neutrophil recruitment. Recruited neutrophils from the
circulation form an abscess that helps control and limit the spread of infection, which is ultimately required for bacterial clearance.
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