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ÔÚ¡°Journal of Cardiovascular Pharmacology¡±Í¶ÁËһƪÎÄÕ£¬ÐÞ¸ÄÒâ¼ûÖ»ÓÐÒ»¸öreviewerµÄ¡£Òâ¼ûÈçÏ£¬ÏëÈôó¼Ò°ïÎÒ·ÖÎöһϡ£
Reviewer #1: This is a beginning of dissection of the pathway involved in the sympathetic nervous system's role in fibrotic expansion in the heart. There are some components that could be added to strengthen the conclusions made here.£¨´Ë´¦µÄÒâ˼ÊÇÈò¹³äʵÑéô£¿¾ßÌåҲû˵²¹³äÄÄ·½ÃæµÄʵÑé°¡£¬¸Ð¾õºÜÃÔ㣩 Additionally, THE PAPER MUST BE REWRITTEN BY A NATIVE ENGLISH SPEAKER FOR PORPER SYNTAX AND GRAMMAR.

1. A study by Hirota et al in lung recently came out looking at lung fibroblasts. It would be interesting to have this discussed in the context of the work you have done in the heart. (Hirota et al, Gene expression profiling of lung myofibroblasts reveals the anti-fibrotic effects of cyclosporine. Tohoku J. of Exp Med 233(4) pg 283-293, 2014).£¨´Ë´¦µÄÒâ˼ÊÇÈÃÒýÓôËÎÄÏ×£¬ÎÒÒѾ­ÒýÓÃÁË£¬µ«ÊÇÔڻظ´Òâ¼ûʱ¸ÃÔõô¿ä½±´ËÎÄÕ£¿×îºÃÄܸø¸öÓ¢ÎİæµÄÄ£°æ£¬±¾ÈËÓ¢Óﲻ̫ºÃ£©

2. AKT, MAPK and ERK have all been shown to be involved in fibrosis and myofibroblast activation. Are they affected by either NE or by inhibition through one of the drugs tested here?
´Ë´¦Ìá¼°µÄAKT, MAPK and ERKÔÚNE´Ì¼¤ÊÇÊÇÓз´Ó¦µÄ£¬ÕâÓÐÎÄÏ×±¨µÀ£¬ÎÒûÓмì²â£¬ÄÇôÎһظ´Òâ¼ûʱÊDz»ÊÇÒª°ÑÈ˼ҵÄÎÄÏ×ÁгöÀ´£¿½âÊÍ×Ô¼ºÃ»ÓÐ×öµÄÔ­Òò£¿

3. Pannexin's have been shown to be important in activation of myofibroblasts in the heart. Are there changes in pannexins in this study?
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