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Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori. Autophagy. 2012 Jul 1;8(7):1045-57 因无法登陆web of science，求SCI文章检索号，填表用，谢谢！ |
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sunshan4379
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linda831223: 金币+5, ★★★★★最佳答案, 十分谢谢！ 2014-08-20 12:03:20
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linda831223: 金币+5, ★★★★★最佳答案, 十分谢谢！ 2014-08-20 12:03:20
jssxh: 金币+5, 检索EPI+1, 谢谢参与，请继续关注本版块！ 2014-08-21 21:26:05
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Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori 作者:Tang, B (Tang, Bin)[ 1,3 ] ; Li, N (Li, Na)[ 1 ] ; Gu, J (Gu, Jiang)[ 1 ] ; Zhuang, Y (Zhuang, Yuan)[ 1 ] ; Li, Q (Li, Qian)[ 1 ] ; Wang, HG (Wang, Hai-Guang)[ 1 ] ; Fang, Y (Fang, Yao)[ 1 ] ; Yu, B (Yu, Bo)[ 1 ] ; Zhang, JY (Zhang, Jin-Yu)[ 1 ] ; Xie, QH (Xie, Qing-Hua)[ 1 ] 更多内容 AUTOPHAGY 卷: 8 期: 7 页: 1045-1057 DOI: 10.4161/auto.20159 出版年: JUL 2012 查看期刊信息 摘要 Helicobacter pylori evade immune responses and achieve persistent colonization in the stomach. However, the mechanism by which H. pylori infections persist is not clear. In this study, we showed that MIR30B is upregulated during H. pylori infection of an AGS cell line and human gastric tissues. Upregulation of MIR30B benefited bacterial replication by compromising the process of autophagy during the H. pylori infection. As a potential mechanistic explanation for this observation, we demonstrate that MIR30B directly targets ATG12 and BECN1, which are important proteins involved in autophagy. These results suggest that compromise of autophagy by MIR30B allows intracellular H. pylori to evade autophagic clearance, thereby contributing to the persistence of H. pylori infections. 关键词 作者关键词:Helicobacter pylori; MIR30B; autophagy; ATG12; BECN1 KeyWords Plus:GASTRIC EPITHELIAL-CELLS; INFLAMMATORY RESPONSE; MONITORING AUTOPHAGY; MICRORNA EXPRESSION; IMMUNE-RESPONSES; CANCER; INDUCTION; INFECTION; RESISTANCE; APOPTOSIS 作者信息 通讯作者地址: Zou, QM (通讯作者) 显示增强组织信息的名称 Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China. 地址: 显示增强组织信息的名称 [ 1 ] Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China 显示增强组织信息的名称 [ 2 ] Chinese Acad Sci, Inst Microbiol, Biol Resource Ctr, Beijing, Peoples R China [ 3 ] Emei Sanat Chengdu Mil Reg, Emeishan, Sichuan, Peoples R China 电子邮件地址:qmzou2007@163.com; mxh95xy@tom.com 基金资助致谢 基金资助机构 授权号 National Natural Science Foundation of China (NSFC) 81071412 查看基金资助信息 出版商 LANDES BIOSCIENCE, 1806 RIO GRANDE ST, AUSTIN, TX 78702 USA 类别 / 分类 研究方向:Cell Biology Web of Science 类别:Cell Biology 文献信息 文献类型:Article 语种:English 入藏号: WOS:000307830600004 ISSN: 1554-8627 期刊信息 Impact Factor (影响因子): Journal Citation Reports® 其他信息 IDS 号: 993BX Web of Science 核心合集中的 "引用的参考文献": 43 Web of Science 核心合集中的 "被引频次": 5 |
2楼2014-08-20 11:53:17
