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北京石油化工学院2026年研究生招生接收调剂公告
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Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori. Autophagy. 2012 Jul 1;8(7):1045-57
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linda831223: 金币+5, ★★★★★最佳答案, 十分谢谢! 2014-08-20 12:03:20
jssxh: 金币+5, 检索EPI+1, 谢谢参与,请继续关注本版块! 2014-08-21 21:26:05
Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori
作者:Tang, B (Tang, Bin)[ 1,3 ] ; Li, N (Li, Na)[ 1 ] ; Gu, J (Gu, Jiang)[ 1 ] ; Zhuang, Y (Zhuang, Yuan)[ 1 ] ; Li, Q (Li, Qian)[ 1 ] ; Wang, HG (Wang, Hai-Guang)[ 1 ] ; Fang, Y (Fang, Yao)[ 1 ] ; Yu, B (Yu, Bo)[ 1 ] ; Zhang, JY (Zhang, Jin-Yu)[ 1 ] ; Xie, QH (Xie, Qing-Hua)[ 1 ] 更多内容
AUTOPHAGY
卷: 8  期: 7  页: 1045-1057
DOI: 10.4161/auto.20159
出版年: JUL 2012
查看期刊信息
摘要
Helicobacter pylori evade immune responses and achieve persistent colonization in the stomach. However, the mechanism by which H. pylori infections persist is not clear. In this study, we showed that MIR30B is upregulated during H. pylori infection of an AGS cell line and human gastric tissues. Upregulation of MIR30B benefited bacterial replication by compromising the process of autophagy during the H. pylori infection. As a potential mechanistic explanation for this observation, we demonstrate that MIR30B directly targets ATG12 and BECN1, which are important proteins involved in autophagy. These results suggest that compromise of autophagy by MIR30B allows intracellular H. pylori to evade autophagic clearance, thereby contributing to the persistence of H. pylori infections.
关键词
作者关键词:Helicobacter pylori; MIR30B; autophagy; ATG12; BECN1
KeyWords Plus:GASTRIC EPITHELIAL-CELLS; INFLAMMATORY RESPONSE; MONITORING AUTOPHAGY; MICRORNA EXPRESSION; IMMUNE-RESPONSES; CANCER; INDUCTION; INFECTION; RESISTANCE; APOPTOSIS
作者信息
通讯作者地址: Zou, QM (通讯作者)
显示增强组织信息的名称        Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China.
地址:
显示增强组织信息的名称        [ 1 ] Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China
显示增强组织信息的名称        [ 2 ] Chinese Acad Sci, Inst Microbiol, Biol Resource Ctr, Beijing, Peoples R China
              [ 3 ] Emei Sanat Chengdu Mil Reg, Emeishan, Sichuan, Peoples R China
电子邮件地址:qmzou2007@163.com; mxh95xy@tom.com
基金资助致谢
基金资助机构        授权号
National Natural Science Foundation of China (NSFC)        
81071412
查看基金资助信息   
出版商
LANDES BIOSCIENCE, 1806 RIO GRANDE ST, AUSTIN, TX 78702 USA
类别 / 分类
研究方向:Cell Biology
Web of Science 类别:Cell Biology
文献信息
文献类型:Article
语种:English
入藏号: WOS:000307830600004
ISSN: 1554-8627
期刊信息
Impact Factor (影响因子): Journal Citation Reports®
其他信息
IDS 号: 993BX
Web of Science 核心合集中的 "引用的参考文献": 43
Web of Science 核心合集中的 "被引频次": 5
非淡泊无以明志,非宁静无以致远
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