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The ¡°vascular normalization¡± hypothesis is a potential resolution of this paradox. The vascular abnormalities in tumors stem from an imbalance between proangiogenic and antiangiogenic factors in the local milieu. The ¡°vascular normalization¡± hypothesis states that direct or indirect antiangiogenic therapy (usually therapy aimed toneutralize VEGF activity) tips the imbalance between pro- and antiangiogenic factors back toward equilibrium.

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