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The phenotype is caused by reduced expression of a ta-siRNA, tasiR-ARF, and overexpression of its targets, the ETT/ARF genes (25¨C27). tasiR-ARF and its target genes are conserved among Arabidopsis, maize, and rice (21, 28). Mutations in LEAF BLADELESS in maize, which encodes a homologue
of SUPRESSOR of GENE SILENCING3 (SGS3) in Arabidopsis and is involved in the ta-siRNA production pathway,result in a similar seedling phenotype to sho1 and sho2, including defects in leaf polarity.The tasiR-ARF cleavage
products of the ETT/ARF genes were also severely reduced in sho mutant seedlings¡£It is also possible that a defect in the production of an unknown ta-siRNA, directed at the precursors of miR166, resulted in increased the stability of the precursors and overaccumulation of miR166.

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