24小时热门版块排行榜    

CyRhmU.jpeg
查看: 338  |  回复: 6
当前主题已经存档。

lhliu2087

银虫 (著名写手)

[交流] 求两篇SCI检索号,每篇3金币

1. Linhua Liu,Yiguo Jiang, Hongyu Zhang, Anne R. Greenlee, Rian Yu and Qiaoyuan Yang. MiR-22 functions as a micro-oncogene in transformed human bronchial epithelial cells induced by anti-benzo[a]pyrene-7,8-diol-9,10-epoxide.Toxicology in vitro.2010; PMID: 20170724

2. Linhua Liu,Yiguo Jiang,Hongyu Zhang,Anne R.Greenlee and Zhiyuan Han.Overexpressed miR-494 down-regulates PTEN gene expression in cells transformed by anti-benzo(a)pyrene-trans-7,8-dihydrodiol-9,10-epoxide.Life Sciences.2010;86(5-6):192-198.
多谢!
回复此楼
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

hzwhut

版主 (著名写手)

优秀版主优秀版主优秀版主优秀版主

第二篇的信息


lhliu2087(金币+1):谢谢参与
作者: Liu LH (Liu, Linhua)1, Jiang YG (Jiang, Yiguo)1, Zhang HY (Zhang, Hongyu)3, Greenlee AR (Greenlee, Anne R.)2, Han ZY (Han, Zhiyuan)1  
来源出版物: LIFE SCIENCES    卷: 86    期: 5-6    页: 192-198    出版年: JAN 30 2010   
被引频次: 0     参考文献: 38     引证关系图      
摘要: Aims: We investigated the functionality of miR-494 in anti-benzo(a)pyrene-trans-7,8-dihydrodiol-9,10-epoxide (anti-BPDE)-transformed human bronchial epithelial cell 16HBE to reveal its potential target coding-gene.
Main methods: The expression of mature miR-494 in cells was detected by miRNA-specific quantitative real-time polymerase chain reaction (QRT-PCR). QRT-PCR and Western blot were used to identify the expression of phosphatase and tensin homolog (PTEN) mRNA and protein. Following activation or inhibition of mature miRNA expression with precursors or antisense inhibitors, PTEN expression, luciferase activities, cell apoptosis, cell growth in soft agar and cell motility were analyzed.

Key findings: The expression of miR-494 increased while PTEN protein appeared to be lower in malignant transformed 16HBE cells. Enforced miR-494 level decreased PTEN protein expression compared to a negative precursor control group. Inhibition of miR-494 expression increased PTEN protein expression compared to negative inhibitor control group. Decreased expression of miR-494 increased caspase-3/7 activities in transformed 16HBE cells, and increased expression of miR-494 decreased this activity. Inhibition of miR-494 also decreased the malignancy of transformed cells.

Significance: MiR-494 regulates the expression of PTEN post-transcriptionally and functions as a micro-oncogene in carcinogenesis induced by anti-BPDE. MiR-494 may be a useful target for gene therapy. (C) 2009 Elsevier Inc. All rights reserved.

文献类型: Article  
语言: English  
作者关键词: Anti-BPDE; Cell transformation; MicroRNA; MiR-494; PTEN  
KeyWords Plus: LUNG-CANCER CELLS; MICRORNA EXPRESSION; PHOSPHORYLATED AKT; GROWTH; PROGNOSIS; CARCINOMA; APOPTOSIS; PROFILES; MIR-21; MIRNAS  
通讯作者地址: Jiang, YG (通讯作者), Guangzhou Med Univ, Inst Chem Carcinogenesis, State Key Lab Resp Dis, 195 Dongfengxi Rd, Guangzhou 510182, Guangdong Peoples R China  
地址:
1. Guangzhou Med Univ, Inst Chem Carcinogenesis, State Key Lab Resp Dis, Guangzhou 510182, Guangdong Peoples R China
2. Jackson Lab, Stem Cell & Primary Cell Facil, Bar Harbor, ME 04609 USA
3. Sun Yat Sen Univ, Dept Med Oncol, Affiliated Hosp 1, Zhuhai 519000, Peoples R China  
电子邮件地址: jiangyiguo@yahoo.com  
基金资助致谢:
基金资助机构 授权号
National Natural Science Foundation of China  30571546
30771780  
Scientific Research Foundation of the State Education Ministry for Returned Overseas Chinese Scholars  2007-24  
Natural Science Foundation of Guangdong Province  07117550
9251018201000004  
Natural Science Key Program of Higher Education Institutions of Guangdong Province, China  06Z021  
Science and Technology Program of Guangzhou Bureau of Education, China  08A093  

[显示基金资助信息]   

This work was supported by the National Natural Science Foundation of China (30571546, 30771780), the Scientific Research Foundation of the State Education Ministry for Returned Overseas Chinese Scholars (2007-24), the Natural Science Foundation of Guangdong Province (07117550, 9251018201000004), the Natural Science Key Program of Higher Education Institutions of Guangdong Province, China (06Z021) and the Science and Technology Program of Guangzhou Bureau of Education, China (08A093).

出版商: PERGAMON-ELSEVIER SCIENCE LTD, THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND  
IDS 号: 552UC  
ISSN: 0024-3205  
DOI: 10.1016/j.lfs.2009.12.002
2楼2010-03-03 17:34:12
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

hzwhut

版主 (著名写手)

优秀版主优秀版主优秀版主优秀版主

第一篇的信息没有查到,是不是才发表的呢
3楼2010-03-03 17:44:51
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

yicaoting

银虫 (正式写手)


lhliu2087(金币+1):谢谢参与
lhliu2087(金币+2): 2010-03-03 20:42
2楼没有给出关键的SCI检索号,完整的信息如下:
FN ISI Export Format
VR 1.0
PT J
AU Liu, LH
   Jiang, YG
   Zhang, HY
   Greenlee, AR
   Han, ZY
AF Liu, Linhua
   Jiang, Yiguo
   Zhang, Hongyu
   Greenlee, Anne R.
   Han, Zhiyuan
TI Overexpressed miR-494 down-regulates PTEN gene expression in cells
   transformed by anti-benzo(a)pyrene-trans-7,8-dihydrodiol-9,10-epoxide
SO LIFE SCIENCES
LA English
DT Article
DE Anti-BPDE; Cell transformation; MicroRNA; MiR-494; PTEN
ID LUNG-CANCER CELLS; MICRORNA EXPRESSION; PHOSPHORYLATED AKT; GROWTH;
   PROGNOSIS; CARCINOMA; APOPTOSIS; PROFILES; MIR-21; MIRNAS
AB Aims: We investigated the functionality of miR-494 in
   anti-benzo(a)pyrene-trans-7,8-dihydrodiol-9,10-epoxide
   (anti-BPDE)-transformed human bronchial epithelial cell 16HBE to reveal
   its potential target coding-gene.
   Main methods: The expression of mature miR-494 in cells was detected by
   miRNA-specific quantitative real-time polymerase chain reaction
   (QRT-PCR). QRT-PCR and Western blot were used to identify the
   expression of phosphatase and tensin homolog (PTEN) mRNA and protein.
   Following activation or inhibition of mature miRNA expression with
   precursors or antisense inhibitors, PTEN expression, luciferase
   activities, cell apoptosis, cell growth in soft agar and cell motility
   were analyzed.
   Key findings: The expression of miR-494 increased while PTEN protein
   appeared to be lower in malignant transformed 16HBE cells. Enforced
   miR-494 level decreased PTEN protein expression compared to a negative
   precursor control group. Inhibition of miR-494 expression increased
   PTEN protein expression compared to negative inhibitor control group.
   Decreased expression of miR-494 increased caspase-3/7 activities in
   transformed 16HBE cells, and increased expression of miR-494 decreased
   this activity. Inhibition of miR-494 also decreased the malignancy of
   transformed cells.
   Significance: MiR-494 regulates the expression of PTEN
   post-transcriptionally and functions as a micro-oncogene in
   carcinogenesis induced by anti-BPDE. MiR-494 may be a useful target for
   gene therapy. (C) 2009 Elsevier Inc. All rights reserved.
C1 [Liu, Linhua; Jiang, Yiguo; Han, Zhiyuan] Guangzhou Med Univ, Inst Chem Carcinogenesis, State Key Lab Resp Dis, Guangzhou 510182, Guangdong, Peoples R China.
   [Greenlee, Anne R.] Jackson Lab, Stem Cell & Primary Cell Facil, Bar Harbor, ME 04609 USA.
   [Zhang, Hongyu] Sun Yat Sen Univ, Dept Med Oncol, Affiliated Hosp 1, Zhuhai 519000, Peoples R China.
RP Jiang, YG, Guangzhou Med Univ, Inst Chem Carcinogenesis, State Key Lab
   Resp Dis, 195 Dongfengxi Rd, Guangzhou 510182, Guangdong, Peoples R
   China.
EM jiangyiguo@yahoo.com
FU National Natural Science Foundation of China [30571546, 30771780];
   Scientific Research Foundation of the State Education Ministry for
   Returned Overseas Chinese Scholars [2007-24]; Natural Science
   Foundation of Guangdong Province [07117550, 9251018201000004]; Natural
   Science Key Program of Higher Education Institutions of Guangdong
   Province, China [06Z021]; Science and Technology Program of Guangzhou
   Bureau of Education, China [08A093]
FX This work was supported by the National Natural Science Foundation of
   China (30571546, 30771780), the Scientific Research Foundation of the
   State Education Ministry for Returned Overseas Chinese Scholars
   (2007-24), the Natural Science Foundation of Guangdong Province
   (07117550, 9251018201000004), the Natural Science Key Program of Higher
   Education Institutions of Guangdong Province, China (06Z021) and the
   Science and Technology Program of Guangzhou Bureau of Education, China
   (08A093).
NR 38
TC 0
PU PERGAMON-ELSEVIER SCIENCE LTD
PI OXFORD
PA THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND
SN 0024-3205
J9 LIFE SCI
JI Life Sci.
PD JAN 30
PY 2010
VL 86
IS 5-6
BP 192
EP 198
DI 10.1016/j.lfs.2009.12.002
PG 7
SC Medicine, Research & Experimental; Pharmacology & Pharmacy
GA 552UC
UT ISI:000274318100007
ER

EF

其中UT ISI:000274318100007,才是唯一的SCI检索号!
4楼2010-03-03 18:00:39
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

yicaoting

银虫 (正式写手)

第一篇尚未被SCI收录,要等一段时间。
5楼2010-03-03 18:02:53
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

tfyihit

木虫 (著名写手)


lhliu2087(金币+1):谢谢参与
3楼ok
6楼2010-03-03 18:44:59
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖

Yorkxu

木虫 (著名写手)


lhliu2087(金币+1):谢谢参与
请问这个号怎么才能得到 ?
7楼2010-03-03 19:49:43
已阅   回复此楼   关注TA 给TA发消息 送TA红花 TA的回帖
相关版块跳转 我要订阅楼主 lhliu2087 的主题更新
普通表情 高级回复(可上传附件)
信息提示
请填处理意见