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ÒÔÏÂÊÇÕⲿ·Ö¶ÎÂ䣺 2. Cell proliferation inhibition induced by cordycepin It is important to understand how cordycepin inhibits cell proliferation in tumor cells in order to develop as a new medicine for cancer prevention and treatment. The A3 adenosine receptor (A3AR) is a member of the AR family; it is overexpressed in cancer and inflammatory cells, however, low expression is exhibited in normal cells (25). Following cordycepin binding to A3AR, G protein is activated and subsequently inhibits the formation of cAMP and indirectly decreases phosphorylation of the serine/threonine kinase glycogen synthase kinase (GSK)‑3¦Â. The resulting increased phosphorylation of ¦Â-catenin causes it to be removed from the cytoplasm by ubiquitination, thereby preventing its nuclear import. A net suppression of cyclin D1 and c‑myc is the result of this, which leads to the inhibition of cell growth (Fig. 1) (26). From above analysis, we may safely draw the conclusion that inhibiting cancer/tumor cell proliferation is one of cordycepin function. ×îºóÕâ¾ä£¬Çë´óÉñÄܲ»Äܰïæд¸ö×ܽᡣ |
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