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yin198716

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Blockade of Dll4-Notch signaling in tumors results in excessive, non-productive angiogenesis with resultant inhibitory effects on tumor growth, even in some tumors that are resistant to anti-VEGF therapies. As Dll4 inhibitors are entering clinical cancer trials, this review aims to provide current perspectives on the function of the Dll4-Notch signaling axis during tumor angiogenesis and as a target for anti-angiogenic cancer therapy.
Oncology
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湖师院学子_

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phu_grassman: 金币+2, thanks for your help. 2013-02-28 10:02:36
sltmac: 金币+3 2013-04-07 13:27:57
DLL4切口肿瘤导致过多的信号封锁,所得对肿瘤生长的抑制作用,非生产性的血管生成,甚至在某些肿瘤,抗VEGF治疗。Dll4的抑制剂进入临床癌症试验,本文旨在对Dll4的Notch信号轴在肿瘤血管生成和抗血管生成治疗癌症的目标函数为当前的观点
2楼2013-02-27 23:01:15
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幻城雪落无声

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sltmac: 金币+7, 翻译EPI+1, 欢迎常来本版交流 2013-04-07 13:28:10
肿瘤中,甚至是一些抗VEGF疗法的肿瘤中,Dll4-Notch 信号的封锁导致过多影响肿瘤生长的受合成抑制的非生产性血管生成。由于Dll4 抑制剂正在进入癌症临床试验,此观点旨在提供在肿瘤血管生成中以 Dll4-Notch 信号为轴的函数的当前观点和作为抗血管生成癌症疗法的一个目标。
3楼2013-03-01 16:24:03
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