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1121prettycat

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In comparison with the well-known SERM, 4-hydroxytamoxifen, AHTN and HHCB have a much lower potency in suppressing estradiol-induced transactivation. Results with various mutant ER receptor types suggest that binding sites of the musk compounds differ from 17beta-estradiol and 4-hydroxytamoxifen. The cell type dependency of ER transactivation shows that caution should be exerted when interpreting effects of estrogenic compounds using in vitro systems. (C) 2002 Elsevier Science (USA).
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1121prettycat

木虫 (正式写手)

The cell type dependency of ER transactivation shows that caution should be exerted when interpreting effects of estrogenic compounds using in vitro systems. 继续求助最后一句
3楼2011-06-17 09:01:41
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fujuan114

禁虫 (小有名气)

【答案】应助回帖

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sltmac(金币+1): 多谢交流~~ 2011-06-16 20:59:29
1121prettycat(金币+10, 翻译EPI+1): 非常感谢 2011-06-17 09:01:20
1121prettycat(金币+10): 全部发放最初求助金币 2011-06-18 09:25:21
与众所周知的SERM比较,4 - 羟基他莫西芬,AHTN和HHCB在抑制雌二醇诱导激活上效价要低得多。各种突变体ER受体类型结果表明,麝香化合物结合位点不同于17β-雌二醇和4 - 羟基他莫西芬。 ER式激活的细胞类型依赖性表明,在说明体外系统使用雌激素化合物的影响时应谨慎。 (C)2002爱思唯尔科技(美国)。
2楼2011-06-16 12:45:00
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