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德国维尔茨堡大学Pedro Friedmann教授课题组招聘岗位制博士/博士后 (细胞生物学)
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各位老师、同学，大家好，德国维尔茨堡大学 José Pedro Friedmann Angeli 教授课题组现招聘 博士后和博士研究生（ERC 项目资助，预计 2026 年初入组，课题组网站 https://www.uni-wuerzburg.de/rvz ... ag-friedmann-angeli）。 课题组主要研究细胞死亡的分子机制，结合 CRISPR 功能基因组学、合成致死和化学生物学等前沿方法。课题组氛围开放、合作性强，拥有先进的基因组学、成像和药物发现平台，鼓励大家开展创新性研究。 如果你对分子生物学、细胞生物学、肿瘤生物学感兴趣，非常欢迎加入我们 ！（申请请提交至pedro.angeli@uni-wuerzburg.de，并抄送至office.friedmann-angeli@uni-wuerzburg.de）更多详情信息如下： Postdoctoral Position & PhD Position in Molecular Cell Biology ERC-funded project in the group of Prof. José Pedro Friedmann Angeli at the Rudolf Virchow Center, Julius-Maximilians-Universität Würzburg, Germany (Link). We invite applications for one Postdoctoral Position and one PhD Position, starting in early 2026, to join our European Research Council (ERC)-funded project - About the Project Our lab investigates the molecular mechanisms of cell survival and cell death, with a strong focus on CRISPR-based functional genomics, synthetic lethal interactions, and chemical biology. This project provides a highly interdisciplinary and collaborative environment, with access to cutting-edge technologies and core facilities. - Postdoctoral Position We seek a highly motivated researcher with: • A PhD (or equivalent) in molecular biology, biochemistry, or a related discipline • Strong expertise in molecular biology techniques (e.g., CRISPR-Cas9, genetic engineering) • Basic bioinformatic skills • Independent project management experience • A strong publication record (relative to career stage) The position is initially offered as a one-year contract, with full support provided to apply for prestigious fellowships such as EMBO, MSCA, and Humboldt. We aim to help you secure independent funding and advance your career; however, if applications are not successful, the contract can be extended to ensure continuity of your research - PhD Position We seek an enthusiastic doctoral candidate with: • A Master’s degree in molecular biology, biochemistry, or a related field • Foundational expertise in molecular biology/biochemistry and in vitro cell culture • High motivation and strong interest in functional genetics and cell death research • Team spirit and curiosity for interdisciplinary science - What We Offer • An excellent scientific environment at the University of Würzburg, a leading biomedical research hub in Germany • A research project addressing a cutting-edge and highly relevant topic in biomedical science • A dynamic, international and diverse research team • Access to advanced facilities for genomics, imaging, and drug discovery • Salary and benefits according to German public service regulations (TV-L) - Application Please send a single PDF including: • CV • Cover letter • Publication list (for postdoc applicants) • Contact details of at least two referees Applications should be submitted to pedro.angeli@uni-wuerzburg.de and cc ́ed to office.friedmann-angeli@uni-wuerzburg.de Review of applications will begin immediately and continue until the positions are filled. - Relevant publications: 1. Skafar V, et al., Riboflavin metabolism shapes FSP1-driven ferroptosis resistance. Biorxiv. 2025. Aug 6; DOI: 10.1101/2025.08.05.668651 2. Chen Z, et al., PRDX6 contributes to selenocysteine metabolism and ferroptosis resistance. Mol Cell. 2024 Dec 5;84(23):4645-4659.e9. DOI: 10.1016/j.molcel.2024.10.027 3. Freitas FP, et al., 7-Dehydrocholesterol is an endogenous suppressor of ferroptosis. Nature. 2024 Feb;626(7998):401-410. DOI: 10.1038/s41586-023-06878-9. 4. Kreß JKC, et al., Cell Rep. 2023 Jul 25;42(7):112724. DOI: 10.1016/j.celrep.2023.112724 5. Alborzinia H, et al., LRP8-mediated selenocysteine uptake is a targetable vulnerability in MYCN-amplified neuroblastoma. EMBO Mol Med. 2023 Aug 7;15(8):e18014. DOI: 10.15252/emmm.202318014 6. Doll S*, Freitas FP*, et al., FSP1 is a glutathione-independent ferroptosis suppressor. Nature. 2019 Nov;575(7784):693-698. doi: 10.1038/s41586-019-1707-0 |
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